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Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation
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Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation

King, Ben C ; Kulak, Klaudia ; Krus, Ulrika ; Rosberg, Rebecca ; Golec, Ewelina ; Wozniak, Katarzyna ; Gomez, Maria F ; Zhang, Enming ; O'Connell, David J ; Renström, Erik ; Blom, Anna M

Cell metabolism, 2019-01, Vol.29 (1), p.202-210.e6 [Peer Reviewed Journal]

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2
Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation
Material Type:
Article
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Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation

King, Ben ; Kulak, Klaudia ; Krus, Ulrika ; Rosberg, Rebecca ; Golec, Ewelina ; wozniak, katarzyna ; Gomez, Maria ; Zhang, Enming ; O'Connell, David J ; Renström, Erik ; Blom, Anna

Cell metabolism, 2019-01-08, Vol.29 (1), p.6 [Peer Reviewed Journal]

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3
Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents [beta] Cell Death via ATG16L1 Interaction and Autophagy Regulation
Material Type:
Article
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Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents [beta] Cell Death via ATG16L1 Interaction and Autophagy Regulation

King, Ben C ; Kulak, Klaudia ; Krus, Ulrika ; Rosberg, Rebecca ; Golec, Ewelina ; Wozniak, Katarzyna ; Gomez, Maria F ; Zhang, Enming ; O'Connell, David J ; Renstrom, Erik ; Blom, Anna M

Cell metabolism, 2019-01-08, Vol.29 (1), p.202 [Peer Reviewed Journal]

Full text available

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